Vigabatrin-Induced Retinal Functional Alterations and Second-Order Neuron Plasticity in C57BL/6J Mice

Chan, K., K. Gibson, S. Gloe, M. Hoon, E. Jansen, J. Kiland, G. McLellan, B. Pattnaik, J. Roullet, G. Salomons, J. Ver Hoeve, K. Vogel, B. Wahlgren, D. Walters, B. Wetherbee, and J. Williams. Vigabatrin-Induced Retinal Functional Alterations and Second-Order Neuron Plasticity in C57BL/6J Mice. Investigative Ophthalmology & Visual Science, 2020, p. Epub PubMed Text.

Abstract

Purpose: Vigabatrin (VGB) is an effective antiepileptic that increases concentrations of inhibitory γ-aminobutyric acid (GABA) by inhibiting GABA transaminase. Reports of VGB-associated visual field loss limit its clinical usefulness, and retinal toxicity studies in laboratory animals have yielded conflicting results.